Epithelial Information and Courses from MediaLab, Inc.
These are the MediaLab courses that cover Epithelial and links to relevant pages within the course.
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| We know that hemostatic events are triggered by a break in the inert epithelial lining of the vasculature, but what specific product(s) act as initiators? | View Page |
| Primary Hemostasis – The Vascular System Our blood circulates freely through undamaged, intact vessels. The design of the vasculature, or blood vessels, is such that the walls of the vessels are chemically inert to both coagulation factors and platelets under normal conditions. Damage to a vessel breaks that inert epithelial lining, exposing the subendothelium and collagen, and releasing chemical signals that trigger subsequent hemostatic mechanisms. | View Page |
| Life Span and function of Eosinophils Eosinophils have a circulating half-life of approximately 18 hours and a tissue life span of at least 6 days.They are capable of locomotion and phagocytosis and can enter inflammatory sites, but do so less readily than neutrophils.In tissues the primary location for eosinophils is in the epithelial barriers to the outside world such as, lungs, skin and GI tract.They are capable of returning to the circulating blood and bone marrow after they enter the tissues. | View Page |
| Renal Epithelial Fragments Renal epithelial fragments of collecting duct origin are composed of three or more cuboidal cells. These fragments indicate a more severe form of renal tubule injury with basement membrane disruption. Proximal and distal convoluted tubule renal epithelial cells are not found in fragment form. In addition to the indication of severe tubule damage, proper identification of these fragments is important to avoid a false positive diagnosis of low-grade transitional cell carcinoma. Transitional cell carcinoma is a type of cancer seen in 71% of cases of malignant tumors of the ureter. | View Page |